Publications
[65] Han R*. (2024) Hit-and-run epigenome editing durably lowers cholesterol in mice. Molecular Therapy doi: 10.1016/j.ymthe.2024.03.027. Full Text
[64] Han R*. (2023) First in vivo base-editing trial shows promise. Molecular Therapy 32: 1-2. Full Text
[63] Li H, Wang P, Zhang C, Zuo Y, Zhou Y, Han R*. (2023) Defective BVES-mediated feedback control of cAMP in muscular dystrophy. Nature Communications 14: 1785. Full Text
[62] Zuo Y, Wang P, Gao Y, Zhang C, Zhou Y, Li H, Chen Z, Yang Z, Xu J, Zhang J, Chen YE, Han R*. (2022) A self-inactivating system for AAV-mediated in vivo base editing. biorxiv doi: 10.21203/rs.3.rs-1663604/v1. Full Text
[61] Koike T, Koike Y, Guo Y, Yang D, Song J, Xu J, Zhao X, Zhu T, Li R, Wen B, Sun D, Rom O, Han R, Fan J, Garcia-Barrio MT, Zhang J, Chen YE. (2022) IDOL deficiency inhibits cholesterol-rich diet-induced atherosclerosis in rabbits. biorxiv doi: 10.1101/2022.05.03.490218. Full Text
[60] Zuo Y, Zhang C, Zhou Y, Li H, Xiao W, Herzog RW, Xu J, Zhang J, Chen EY, Han R*. (2023) Liver-specific in vivo base editing of Angptl3 via AAV delivery efficiently lowers blood lipid levels in mice. Cell & Bioscience 13: 109. Full Text
[59] Wang P, Li H, Zhu M, Han RY, Guo S, Han R*. (2022) Correction of DMD in human iPSC-derived cardiomyocytes by base editing-induced exon skipping. Molecular Therapy - Methods & Clinical Development 28: 40-50. Full Text
[58] Li H, Wang P, Hsu E, Pinckard KM, Stanford KI and Han R*. (2022) Systemic AAV9.BVES Delivery Ameliorates Muscular Dystrophy in a Mouse Model of LGMDR25. Molecular Therapy doi: 10.1016/j.ymthe.2022.11.012. Full Text
[57] Zou X, Ouyang H, Lin F, Zhang H, Yang Y, Pang D, Han R*, Tang X*. (2022) MYBPC3 deficiency in cardiac fibroblasts drives their activation and contributes to fibrosis. Cell Death & Disease doi: 10.1038/s41419-022-05403-6. Full Text
[56] Li H, Zhang L, Zhang L and Han R*. (2022) Autophagy in striated muscle diseases Frontiers in Cardiovascular Medicine doi: 10.3389/fcvm.2022.1000067. Full Text
[55] Zhang M, Urabe G, Ozer HG, Xie X, Webb A, Shirasu T, Li J, Han R, Kent KC, Wang B and Guo LW. (2022) Angioplasty induces epigenomic remodeling in injured arteries. Life Science Alliance 5: e202101114. Full Text
[54] Li H, Xu L, Gao Y, Zuo Y, Yang Z, Zhao L, Chen Z, Guo J, Han R*. (2021) BVES Is a Novel Interactor of ANO5 and Regulates Myoblast Differentiation. Cell & Bioscience 11(1): 222. Full Text
[53] Li H, Lin P-H, Pranav G, Li X, Zhao L, Zhou X, Li Z, Wei S, Xu L, Han R, Lu J, Tan T, Yang D-H, Chen Z-S, Pawlik TM, Merritt RE, Ma J (2021) MG53 suppresses tumor progression and stress granule formation by modulating G3BP2 activity in non-small cell lung cancer. Molecular Cancer 20: 118. Full Text
[52] Zou X, Ouyang H, Pang D, Han R* and Tang X*. (2021) Pathological alterations in the gastrointestinal tract of a porcine model of DMD. Cell & Bioscience 11(1): 131. Full Text
[51] Xu L, Zhang C, Li H, Wang P, Gao Y, Mokadam NA, Ma J, Arnold WD, Han R*. (2021) Efficient precise in vivo base editing in adult dystrophic mice. Nature Communications 12(1): 3719. Full Text
[50] Yang D, Liang X, Pallas B, Hoenerhoff M, Ren Z, Han R, Zhang J, Chen YE, Jin J-P, Sun F, Xu J. (2021) Production of CFTR-deltaF508 Rabbits Frontiers in Genetics 11: 627666. Full Text
[49] Xu J, Zhang J, Yang D, Song J, Pallas B, Zhang C, Hu J, Peng X, Christensen N, Han R* and Chen YE*. (2021) Gene editing in rabbits: unique opportunities for translational biomedical research. Frontiers in Genetics 12: 642444. Full Text
[48] Li H, Zhao L, Lau YS, Zhang C, Han R*. (2020) Genome-wide CRISPR screen identifies LGALS2 as an oxidative stress-responsive gene with an inhibitory function on colon tumor growth. Oncogene doi: 10.1038/s41388-020-01523-5. Full Text
[47] Lau YS, Zhao L, Zhang C, Li H, Han R*. (2020) Genetic disruption of the inflammasome adaptor ASC has minimal impact on the pathogenesis of Duchenne muscular dystrophy in mdx Mice. Life Sciences 257: 118069. Full Text
[46] Sui T, Xu L, Lau YS, Liu D, Liu T, Gao Y, Lai L, Han R, Li Z. (2020) Correction: Development of muscular dystrophy in a CRISPR-engineered mutant rabbit model with frame-disrupting ANO5 mutations. Cell Death Dis doi: 10.1038/s41419-020-2659-x: . Full Text
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